Progranulin-mediated deficiency of cathepsin D results in FTD and NCL-like phenotypes in neurons derived from FTD patients

Valdez, Clarissa; Wong, Yvette C; Schwake, Michael; Bu, Guojun; Wszolek, Zbigniew K; Krainc, Dimitri

Progranulin-mediated deficiency of cathepsin D results in FTD and NCL-like phenotypes in neurons derived from FTD patients

Valdez C, Wong YC, Schwake M, Bu G, Wszolek ZK, Krainc D (2017)
Human Molecular Genetics 26(24): 4861-4872.

Zeitschriftenaufsatz | Veröffentlicht | Englisch

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DOI

https://doi.org/10.1093/hmg/ddx364

Autor*in

Valdez, Clarissa; Wong, Yvette C; Schwake, Michael^UniBi ; Bu, Guojun; Wszolek, Zbigniew K; Krainc, Dimitri

Einrichtung

Fakultät für Chemie > Biochemie III

Erscheinungsjahr

2017

Zeitschriftentitel

Human Molecular Genetics

Band

26

Ausgabe

24

Seite(n)

4861-4872

ISSN

0964-6906

eISSN

1460-2083

Page URI

https://pub.uni-bielefeld.de/record/2953334

Zitieren

Valdez C, Wong YC, Schwake M, Bu G, Wszolek ZK, Krainc D. Progranulin-mediated deficiency of cathepsin D results in FTD and NCL-like phenotypes in neurons derived from FTD patients. Human Molecular Genetics. 2017;26(24):4861-4872.

Valdez, C., Wong, Y. C., Schwake, M., Bu, G., Wszolek, Z. K., & Krainc, D. (2017). Progranulin-mediated deficiency of cathepsin D results in FTD and NCL-like phenotypes in neurons derived from FTD patients. Human Molecular Genetics, 26(24), 4861-4872. https://doi.org/10.1093/hmg/ddx364

Valdez, Clarissa, Wong, Yvette C, Schwake, Michael, Bu, Guojun, Wszolek, Zbigniew K, and Krainc, Dimitri. 2017. “Progranulin-mediated deficiency of cathepsin D results in FTD and NCL-like phenotypes in neurons derived from FTD patients”. Human Molecular Genetics 26 (24): 4861-4872.

Valdez, C., Wong, Y. C., Schwake, M., Bu, G., Wszolek, Z. K., and Krainc, D. (2017). Progranulin-mediated deficiency of cathepsin D results in FTD and NCL-like phenotypes in neurons derived from FTD patients. Human Molecular Genetics 26, 4861-4872.

Valdez, C., et al., 2017. Progranulin-mediated deficiency of cathepsin D results in FTD and NCL-like phenotypes in neurons derived from FTD patients. Human Molecular Genetics, 26(24), p 4861-4872.

C. Valdez, et al., “Progranulin-mediated deficiency of cathepsin D results in FTD and NCL-like phenotypes in neurons derived from FTD patients”, Human Molecular Genetics, vol. 26, 2017, pp. 4861-4872.

Valdez, C., Wong, Y.C., Schwake, M., Bu, G., Wszolek, Z.K., Krainc, D.: Progranulin-mediated deficiency of cathepsin D results in FTD and NCL-like phenotypes in neurons derived from FTD patients. Human Molecular Genetics. 26, 4861-4872 (2017).

Valdez, Clarissa, Wong, Yvette C, Schwake, Michael, Bu, Guojun, Wszolek, Zbigniew K, and Krainc, Dimitri. “Progranulin-mediated deficiency of cathepsin D results in FTD and NCL-like phenotypes in neurons derived from FTD patients”. Human Molecular Genetics 26.24 (2017): 4861-4872.

Daten bereitgestellt von European Bioinformatics Institute (EBI)