Whole-genome paired-end analysis confirms remarkable genomic stability of atypical teratoid/rhabdoid tumors

Hoell JI, Gombert M, Bartenhagen C, Ginzel S, Husemann P, Felsberg J, Reifenberger G, Eggert A, Dugas M, Schönberger S, Borkhardt A, et al. (2013)
Genes, chromosomes & cancer 52(10): 983-985.

Zeitschriftenaufsatz | Veröffentlicht | Englisch
 
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DOI
Autor*in
Hoell, Jessica I; Gombert, Michael; Bartenhagen, Christoph; Ginzel, Sebastian; Husemann, PeterUniBi; Felsberg, Jörg; Reifenberger, Guido; Eggert, Angelika; Dugas, Martin; Schönberger, Stefan; Borkhardt, Arndt; Fischer, Ute
Alle
Einrichtung
Centrum für Biotechnologie
Technische Fakultät
Erscheinungsjahr
2013
Zeitschriftentitel
Genes, chromosomes & cancer
Band
52
Ausgabe
10
Seite(n)
983-985
ISSN
1045-2257
Page URI
https://pub.uni-bielefeld.de/record/2621151

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Hoell JI, Gombert M, Bartenhagen C, et al. Whole-genome paired-end analysis confirms remarkable genomic stability of atypical teratoid/rhabdoid tumors. Genes, chromosomes & cancer. 2013;52(10):983-985.
Hoell, J. I., Gombert, M., Bartenhagen, C., Ginzel, S., Husemann, P., Felsberg, J., Reifenberger, G., et al. (2013). Whole-genome paired-end analysis confirms remarkable genomic stability of atypical teratoid/rhabdoid tumors. Genes, chromosomes & cancer, 52(10), 983-985. doi:10.1002/gcc.22092
Hoell, Jessica I, Gombert, Michael, Bartenhagen, Christoph, Ginzel, Sebastian, Husemann, Peter, Felsberg, Jörg, Reifenberger, Guido, et al. 2013. “Whole-genome paired-end analysis confirms remarkable genomic stability of atypical teratoid/rhabdoid tumors”. Genes, chromosomes & cancer 52 (10): 983-985.
Hoell, J. I., Gombert, M., Bartenhagen, C., Ginzel, S., Husemann, P., Felsberg, J., Reifenberger, G., Eggert, A., Dugas, M., Schönberger, S., et al. (2013). Whole-genome paired-end analysis confirms remarkable genomic stability of atypical teratoid/rhabdoid tumors. Genes, chromosomes & cancer 52, 983-985.
Hoell, J.I., et al., 2013. Whole-genome paired-end analysis confirms remarkable genomic stability of atypical teratoid/rhabdoid tumors. Genes, chromosomes & cancer, 52(10), p 983-985.
J.I. Hoell, et al., “Whole-genome paired-end analysis confirms remarkable genomic stability of atypical teratoid/rhabdoid tumors”, Genes, chromosomes & cancer, vol. 52, 2013, pp. 983-985.
Hoell, J.I., Gombert, M., Bartenhagen, C., Ginzel, S., Husemann, P., Felsberg, J., Reifenberger, G., Eggert, A., Dugas, M., Schönberger, S., Borkhardt, A., Fischer, U.: Whole-genome paired-end analysis confirms remarkable genomic stability of atypical teratoid/rhabdoid tumors. Genes, chromosomes & cancer. 52, 983-985 (2013).
Hoell, Jessica I, Gombert, Michael, Bartenhagen, Christoph, Ginzel, Sebastian, Husemann, Peter, Felsberg, Jörg, Reifenberger, Guido, Eggert, Angelika, Dugas, Martin, Schönberger, Stefan, Borkhardt, Arndt, and Fischer, Ute. “Whole-genome paired-end analysis confirms remarkable genomic stability of atypical teratoid/rhabdoid tumors”. Genes, chromosomes & cancer 52.10 (2013): 983-985.

2 Zitationen in Europe PMC

Daten bereitgestellt von Europe PubMed Central.

Phosphoproteomic analysis reveals Smarcb1 dependent EGFR signaling in Malignant Rhabdoid tumor cells.
Darr J, Klochendler A, Isaac S, Geiger T, Eden A., Mol Cancer 14(), 2015
PMID: 26370283
Molecular pathways: SWI/SNF (BAF) complexes are frequently mutated in cancer--mechanisms and potential therapeutic insights.
Wang X, Haswell JR, Roberts CW., Clin Cancer Res 20(1), 2014
PMID: 24122795

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